Chapter 27 - Fluid. Electrolyte and pH Balance

 

(figures relate to Tortora/Grabowski 9th edition of Principles of Anatomy and Physiology)

 

1/3 of all body fluid is ECF (major ions present are Na+ and Cl-)

2/3 of all body fluid is ICF (major ions present are K+ and HPO42-)

80% of the ECF is interstitial fluid,   20% is plasma

We gain water, daily, via: (#l) ingested (moist) foods and liquids, (#2) metabolic reactions

We lose water, daily, via: GI tract, urinary tract, lungs, skin

Figs: 27.3 and 27.4 summarize the regulatory pathways we discussed in Ch. 26

Enemas can cause electrolyte imbalances (and even death) if a fluid is used which is too hypotonic or hypertonic to our body fluids.

Edema is caused by retention of Na+ and therefore, H2O - due to renal failure and/or too much aldosterone secretion.

People at risk for electrolyte imbalances: those that can't communicate thirst impulses, severe trauma/burn patients (pts), elderly, infants, hospitalized pts., people receiving IV's, catheters or diuretics, people experiencing excessive fluid loss or requiring lots of fluids.

Blood Imbalances:

1 - hypernatremia - high sodium: due to high salt diet, hypertonic IV, water deprivation

or dehydration

Symptoms: edema due to water moving into ECF from cells, this also causes excessive thirst, hypertension, convulsions

2 - hyperkalemia - high potassium: due to high intake, low aldosterone (low Na reabs.)

Symptoms: nausea, vomiting, diarrhea, ventricular fibrillation (may cause death)*, metabolic acidosis

3 - hypokalemia - low potassium: excessive vomiting/diarrhea, high aldosterone, diuretics

Symptoms: muscle fatigue, mental confusion, increased urine output, shallow respirations, changes in EGG

4 - hypercalcemia - high calcium: due to hyperparathyroidism or high Vit. D intake

Symptoms: nausea, vomiting, anorexia, itching, depression

5 - hypocalcemia — low calcium: due to hypoparathyroidism or increased Ca loss

 

symptoms: muscle cramps, tetany, bone fractures, convulsions, spasms


Metabolic Acidosis (plasma HCO3- reduced 50% and pH drops below 7.35)

 Due to: NH4Cl ingestion, diabetic ketosis, loss of bicarbonate from diarrhea

Compensation:

1 - Increase respiration (hyperventilation) leads to increase CO2 loss, which leads to decrease

carbonic acid and H+

2  - Kidney increase H+ excretion (returns 1 HCO3-, a base, to plasma)

3  - Ammonia and phosphate buffers H+ in collecting duct

Metabolic Alkalosis (plasma HCO3- increased and pH rises above 7.45)

 Due to: Prolonged vomiting, overuse of diuretics (loss of H+)

Compensation:

1  - Decrease respiration (hypoventilation) leads to increase Pco2 plasma, which leads to
increased carbonic acid and H+ in plasma

2  – Kidney increase HCO3- excretion (a base)

Respiratory Acidosis (increase PCO2 plasma and pH drops below 7.35)

Due to: Hypoventilation due to emphysema or airway obstructions

Compensation:

1  - The increased PCO2 in plasma causes all cells to become more acidic (higher H+)

2  - Kidney increase H+ excretion (returns 1 HCO3-, a base, to plasma)